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Targeting human endothelial cells with glutathione and alanine increases the crossing of a polypeptide nanocarrier through a bloodbrain barrier model and entry to human brain organoids

Under pathological conditions, when excessive glutamate is released from the presynaptic membrane or the reuptake function of glutamate is impaired, the extracellular glutamate concentration increases

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However, it is noteworthy that, while upregulation of glutamine metabolism is a phenomenon present in many cancer cells, recent studies have shown a decrease in glutamine metabolism in cholangiocarcinoma, glioblastoma multiforme, liver hepatocellular carcinoma, and thyroid carcinoma, demonstrating tissue heterogeneity in glutamine metabolism [40]