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Laboratory studies demonstrate that senescent cancer cells maintain similar FOXO4-p53 interactions that protect them from apoptosis, making them susceptible to FOXO4-DRI-mediated elimination while providing researchers with tools for studying cancer cell senescence and survival mechanisms

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Silvin et al., 2022) in human specimen and translational murine models (Figure 1, Table 1)

Furthermore, accelerated glucose depletion alleviates T cell suppression and promotes the repolarization of immunosuppressive M2 macrophages towards antitumor M1 phenotypes, ultimately reversing tumor immunosuppression [75, 76]

In addition, ROS generated in mitochondria and the endoplasmic reticulum also contribute to the development of hypertension (157, 158)

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