Zinc cooperates with p53 to inhibit the activity of mitochondrial aconitase through reactive oxygen species accumulation
Shah and colleagues analyzed ten small clinical studies (with a maximum of 40 participants each), five of which included patients with sepsis
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Understanding these causes can help manage the symptoms and prevent future occurrences
Reported molecular endpoints include VEGFR2 expression and internalisation, FAK and paxillin phosphorylation, growth hormone receptor expression in tendon fibroblasts, and Egr-1 pathway activity
