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Groop P-H, Cooper ME, Perkovic V, Emser A, Woerle H-J, von Eynatten M

This initial evidence highlights a promising avenue for extending the application of YSTJ into AD research, addressing key pathological mechanisms such as neuroinflammation and synaptic dysfunction

Our results therefore confirm that P53 act synergistically to regulate m6A methylation via the YTHDF family in cardiomyocytes

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Understanding the evidence, safety considerations, and lack of proven synergistic benefit is essential for informed clinical decision-making when patients consider combining these treatments