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The phosphorylated SMAD2/3 (pSMAD2/3) then forms a complex with SMAD4,[66] which translocates to the nucleus to regulate gene expression, thereby negatively regulating myoblast proliferation,[14] differentiation, and fusion.[67] MSTN signaling can be inhibited by (1) blocking the interaction between the active C-terminal dimer and ActRIIA/B, as seen with anti-MSTN antibodies such as MYO-029 and landogrozumab
The injection site should be rotated each week
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Nutrients, 15 (11), 2433
Each new indication opens another trial program
This measure can ease the adverse effects while retaining some therapeutic advantages
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