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These mucosal regenerative effects have been consistently demonstrated in experimental animal models of IBD (87), complementing their currently approved use of GLP-2 analogs in short bowel syndrome (88, 89)

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These studies have been enabled by the discovery of novel small molecules, including Fer-1 [5], improved Fer-1 analogs [7, 8] and liproxstatin-1 [9], that potently and specifically block ferroptosis

Consistent with these findings, the current study confirmed that the semaglutide-intensified treatment group exhibited reduced fasting plasma glucose, fasting insulin, total cholesterol, triglyceride, and LDL-C levels compared with baseline

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