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Nicotine-induced VEGF production was mediated by nAChRs via activation of the VEGF and its receptor as well as the extracellular signal-regulated kinase (ERK)1/2 pathway [27]

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Autophagy is an evolutionarily conserved process for degradation and recycling of cellular components in response to various forms of stress and known to take place in damage control for mitochondria

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