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In the case of eNOS uncoupling due to relative intracellular BH 4 deficiency, this phenomenon could be prevented by simultaneous overexpression of the BH 4 synthesizing enzyme GTP cyclohydrolase I, which has been demonstrated in isolated cells 92 and in atherosclerotic animal models

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Additionally, other types of NAMPT activators, such as NAT-5r and SBI-797812, have demonstrated effectiveness in a mouse model for CIPN, which may indicate their therapeutic promise in cognitive disorders 224 (Fig

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