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The peptide reduces tumor necrosis factor-alpha (TNF-) and interleukin-6 (IL-6) expression in gastric tissue exposed to indomethacin, a known ulcerogen

The inflammatory milieu subsequently drives hepatic stellate cell activation and transdifferentiation into myofibroblasts, leading to excessive extracellular matrix deposition and progressive fibrosis, which represents the primary determinant of long-term liver-related outcomes [17]

The combination therapy also provides patient satisfaction and displays effectiveness, yielding excellent cosmetic results

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