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Notably, the IL-6/JAK2/STAT3 pathway exhibits bidirectional regulation: it promotes the accumulation of PUFAs by activating fatty acid synthase (FASN), thereby increasing the risk of ferroptosis, while simultaneously inhibiting CRT exposure to weaken the immunogenicity of ICD (Johnson et al., 2020) (201)

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Research ArticleCell biologyImmunology Open Access | 10.1172/jci.insight.196167 1 Der Simulierte Mensch, a science framework of Technische Universitt Berlin and Charit Universittsmedizin Berlin, Berlin, Germany

Changes in the subjects functional status (ADL and IADL) were not observed, implying that the effects on ADL and IADL were not significant