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It was shown that the deletion of E155 in GSTO1-1 has a significant effect on enzymatic stability and may consequently alter the physiologic role of the enzyme in the brain regions 53
Most patients have a chronic persistent form of the disease, as full remission is uncommon and sustained remission is even more unlikely to occur
Here, we have utilized liver-specific overexpression of cation transport regulator homolog 1 ( Chac1 ), a recently identified intracellular GSH-degrading enzyme, to induce hepatic GSH depletion during MASLD progression
Vic theo di phn ng c th quan trng hn l ko di thi gian dng mt cch my mc
Oncogene 22 , 72657279 (2003)