cigarette smoke mitochondrial depolarization lung epithelial cells SS-31 mitigates oxidative stress and restores function in smoke-damaged oral via PINK1-mediated mitophagy Diagram representing the initiation of

RESEARCH PAPER Cardiovascular functions and arterial stiffness after JUUL use More details Hide details 1 Department of Psychiatry and Psychotherapy, Klinikum der Universitt Mnchen, Munich, Germany 2 Medizinische Klinik III, Universittsklinikum Schleswig-Holstein, Lbeck, Germany 3 Airway Research Center North, Germany 4 Department of Chemical and Product Safety, German Federal Institute for Risk Assessment, Berlin, Germany 5 Institute of Pharmacy, Department of Biology, Chemistry and Pharmacy, Freie Universitt Berlin, Berlin, Germany 6 Cardiology Kiel, Kiel, Germany Submission date: 2021-08-08 Final revision date: 2021-10-31 Acceptance date: 2021-11-28 Publication date: 2022-04-01 Corresponding author Klaas F

In addition to this, nicotine is a risk factor for cardiovascular disease through its well-known effects on endothelial function and stimulation of inflammatory markers such as C-reactive protein 67,68

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