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Glucagon-receptor agonism raises energy expenditure and mobilizes fat, which could in theory favor fat loss over muscle loss

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Exaggerated secretion of glucagon-like peptide-1 (GLP-1) could cause reactive hypoglycemia

Importantly, intravenous infusion of post-prandial levels of GIP increases plasma GLP-1 levels twofold, independent of blood glucose levels (Roberge and Brubaker, 1993), suggesting that GIP stimulates early GLP-1 secretion in response to duodenal luminal nutrients

Classification of RCDs is driven by the underlying molecular machinery and related molecular events linked to a particular RCD