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There might be other pathways/genes account for the differences

In the brain, GHK-Cu has shown promise in animal models: protecting memory under sleep deprivation, producing rapid anti-anxiety effects, and reducing stress-induced damage

Because it shares pharmacological features with licensed GLP-1 receptor agonists such as semaglutide and tirzepatide, clinically relevant interactions including with antidiabetic medicines, anticoagulants, and narrow therapeutic index drugs can be anticipated

The expectation, based on animal data, is faster return to functional loading capacity and better quality of the regenerated tissue rather than simply faster scar formation