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Yes, cigarette smoking accelerates the progression of existing fatty liver disease and increases the risk of developing advanced liver scarring (fibrosis)

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A growing number of studies examining how dysbiosis might drive NAFLD have identified a number of plausible mechanisms, including changes in short-chain fatty acids (SCFAs) metabolism, increased intestinal permeability and lipopolysaccharides (LPS) activation of toll-like receptors (TLRs) and inflammasomes, endogenous ethanol production, decreased choline availability, and trimethylamine production (69)

doi:10.3816/CLC.2004.n.016

In fact, emerging data suggest that hepatocyte apoptosis plays a key component in the progression of simple steatosis to NASH (37)

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