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As the rate-limiting enzyme in UA synthesis, XO activity is modulated by genetic factors (e.g., HPRT1 mutations) and dietary purine intake

This is attributed to its ability to stimulate the production of growth factors, promote angiogenesis (the formation of new blood vessels), and elevate the levels of other proteins that support tissue regeneration

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Iron metabolism and lipid peroxidation signaling are increasingly recognized as central mediators of ferroptosis