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doi: 10.3389/fphar.2024.1447677 264 VieiraEFRamirezPCorreiaADelerue-MatosC

ANO1: more than just calcium-activated chloride channel in cancer

This work was supported by the National Natural Science Foundation of China (U19A2013), the Innovation and Entrepreneurship Talent Funding Project of Jilin Province (2022ZY10), and Technology Development Plan Project of Jilin Province (YDZJ202301ZYTS136)

NF-kappaB Pathway Inhibition Targeted Anti-Inflammatory Control KPV provides potent anti-inflammatory effects through direct inhibition of nuclear factor-kappa B (NF-kappaB), distinguishing KLOW from GLOW through enhanced inflammation control[3]: Competitive blocking of importin-alpha3 interaction with NF-kappaB p65/RelA subunit Prevention of NF-kappaB nuclear translocation in activated cells Inhibition of pro-inflammatory gene transcription Reduction of cytokine production including TNF-alpha, IL-1beta, and IL-6 PepT1-mediated cellular uptake enabling intracellular anti-inflammatory action This mechanism operates at nanomolar concentrations and works through a melanocortin receptor-independent pathway, providing inflammation control without hormonal side effects while complementing the other components regenerative actions

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