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It also has been described that -glutamylcysteine, the intermediate of GSH biosynthesis, is able to act as a Gpx1 cofactor in mitochondrial H 2 O 2 detoxification, mimicking the physiological properties of GSH (Quintana-Cabrera et al., 2012)

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Oleoylethanolamide alleviates hyperlipidaemia-mediated vascular calcification via attenuating mitochondrial DNA stress triggered autophagy-dependent ferroptosis by activating PPARalpha

Ramos, E

Although its primary mechanism was microbiota modulation, the resulting decrease in gut inflammation and oxidative stress was partly attributed to its reactive oxygen species-scavenging action and maintenance of mitochondrial function in epithelial cells