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Rehfeld JF, Friis-Hansen L, Goetze JP, et al

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Simultaneously, aging astrocytes exhibit intrinsic mitochondrial dysfunction that can activate the cGASSTINGYY1 pathway, further amplifying neuroinflammation [92]

Although there is exploration of indirectly reflecting ferroptosis activity by detecting lipid peroxidation products such as MDA and 4-HNE, iron metabolism indicators, or GPX4 expression in joint fluid (Xia S