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This activation commands the cells to immediately upregulate the production of new mitochondria (mitochondrial biogenesis) and shift their metabolism toward the efficient oxidation of fatty acids

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Urinary excretion of N-acetyl-S-allyl-L-cysteine upon garlic consumption by human volunteers

The excessive production of ROS leads to tissue injury which in turn lead to inflammation

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Its primary mechanisms are: Actin regulation critical for cell migration and tissue repair Anti-inflammatory signalling through the NF-B pathway19 Angiogenesis promoting new vessel formation in damaged tissue20 Cardioprotection demonstrating direct myocardial repair properties in animal models21 TB-500 Cardiovascular Long COVID: The Strongest Use Case Long COVID cardiovascular sequelae are well-documented: Myocarditis and pericarditis in a subset of patients POTS (postural orthostatic tachycardia syndrome) Reduced VO max and exercise intolerance Microclotting and endothelial damage TB-500's most compelling evidence base is cardiovascular