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Inhibiting de novo lipogenesis identifies a therapeutic vulnerability in therapy-resistant colorectal cancer

The activated lipase breaks down stored triglycerides into free fatty acids that can be released into the bloodstream and oxidized for energy

The hydroxyl radicals produced via copper-catalyzed Fenton reactions mediate oxidative damage, primarily by oxidizing LDL into oxidized LDL (ox-LDL), which serves as a key driver of atherosclerotic plaque formation [121]

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Druker, B

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