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ZmMYB-CC10 was also shown to activate the expression of ZmAPX4 by binding directly to its promoter using yeast one-hybrid crosses and luciferase assays (Zhang et al., 2022a)

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These models offer powerful tools for identifying latent disease subtypes, modeling progression trajectories, and predicting therapeutic responses, capabilities that are crucial for personalizing neuroprotective strategies [523, 590592]

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