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A study has found miR-16 to be elevated in MASLD patients with simple steatosis and others have found that serum miR-16 is elevated in MASH patients and is associated with the stage of fibrosis (106)

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Mechanistically, activation of GLP-1R by GLP-1 or exenatide phosphorylates STAT3, which drives human monocyte-derived macrophages to develop an M2 phenotype marked by increased expression of CD163, CD204, and IL-10 [86]