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Functionally, this predicted cleavage site is only 5 residues from the C terminal of TXNRD1, and would result in removal of the C-terminal redox center of TXNRD1 (AGCUG), making the enzyme incapable of regenerating reduced thioredoxin

If one takes an external source of HGH it will have to be converted in the liver to IGF-1

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Instead, they rely on trypanothione and trypanothione reductase to protect them from oxidative stress