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Advanced MASH-related liver fibrosis is typically the result of a chronic, sustained inflammatory response involving multiple factors, characterized by abnormal deposition of type I collagen in the extracellular matrix (ECM), with the activation of HSCs being a key driver of liver fibrosis (Chan et al

Repeat injections are typically reserved for patients who experience substantial improvement from the initial injection

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Subsequent studies found that selenium deficiency enhances the aminoglycoside-mediated misincorporation of amino acids at the selenocysteine-specific UGA-codon [169]

H., Santos, H

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