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While GSH is commonly recognized as a component of antioxidant defense systems, it has wide range of roles in disulfide bond formation, detoxification of xenobiotics, metabolite transport, formation of iron-sulfur clusters and cellular signaling

This segment delves into the function of the HGF/c-MET pathway and its subsequent routes in the TME post-activation, encapsulating its impact on various TME cells, as detailed in Table 1