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Levey AS, Schoolwerth AC, Burrows NR, Williams DE, Stith KR, McClellan W

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Similarly, AtPDF2.6 overexpression also enhanced Cd tolerance, while its knockout increased Cd sensitivity, emphasizing its role in detoxification via chelation and efflux (Luo et al., 2019)

There is no FDA-approved combination of the two, but there are no known direct drug interactions either

As shown in Table 2, LoF of ALDH3A2, ALDH4A1, ALDH6A1, ALDH7A1, or ALDH18A1 results in intellectual disabilities, SjgrenLarsson syndrome, hyperprolinemia type II, -hydroxybutyric aciduria (also known as SSADH deficiency), developmental delay, or atypical B6-dependent seizures, respectively