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In summary, our in vitro results suggest, that TGF- and oxidative stress contribute to increased GPX3 expression in lung fibrosis in a cell-type-specific manner, upregulating GPX3 in bronchial epithelial cells and interstitial fibroblasts, respectively

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Subsequently, sections were stained with hematoxylin as counterstaining

Hepatology 45 , 11181128 (2007)

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Getting it right from the outset protects both comfort and consistency, and consistency is what allows GHK-Cu s cumulative biological effects to develop fully