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It's involved in the synthesis of myelin, which is the protective coating around the nerves

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Functional validation using receptor-deficient models confirms that 3-adrenergic signaling is required for these effects, as disruption of this pathway attenuates peptide-induced lipolysis

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This is largely due to altered TJ protein distribution promoting the paracellular leakage of luminal contents (Lerner and Matthias 2015a)

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