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Rodent models, in particular, raise concerns due to their limited translational relevance to human physiology

GxG analysis highlights a buffering interaction between the iron transporter SLC25A37 (A37) and the poorly characterized SLC25A39 (A39)

eNOS, endothelial nitric oxide synthase

iASPP suppresses Gp78-mediated TMCO1 degradation to maintain Ca2 + homeostasis and control tumor growth and drug resistance

Prolonged damage disrupts the balance between collagen secretion and degradation, gradually accumulating to form fibrotic scars, leading to progressive liver fibrosis and impairing liver function