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Related to this, several loss-of-function mutations in genes known to be associated with early onset PD (e.g., PARK2 ) are directly linked to proteopathic stress and compromised mitochondrial functions [44]
And they use some of the same FDA-registered facilities that the drug manufacturers use, Brunner says
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Toxicology and Applied Pharmacology 304: 121132
The Controversial Role of Homocysteine in Neurology: From Labs to Clinical Practice
The dual-incretin logic behind GLP-1 and GIP receptor synergy is also a distinctive marker
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