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Pickering et al., 2006) and to reduce the number of granule cells (Oreland et al., 2010)

The patients presentation aligns well with reported literature on hyperthyroidism-induced hepatic injury, particularly the combination of elevated liver enzymes, severe hyperbilirubinemia, and profound metabolic derangements ( Firstly, the patients autonomous discontinuation of antithyroid medications complicates the clinical picture

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SLC6A14 depletion contributes to amino acid starvation to suppress EMT-induced metastasis in gastric cancer by perturbing the PI3K/AKT/mTORC1 pathway

CXCL10 was previously shown to increase the efficacy of cancer immunotherapy 33

Kava hepatotoxicity in traditional and modern use: The presumed Pacific kava paradox hypothesis revisited