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Additionally, we identify the FHA domain of SNIP1 as a reader of P-SF3B1, guiding the recruitment and assembly of the RES complex during spliceosome activation, and show that mutations in the FHA domain, including the neurodevelopmental disorder-linked E366G variant, impair this interaction, resulting in defects in splicing and cellular proliferation

1 and 2), we assayed whether these effects regulated the proinflammatory response triggered by 25HC

For high-resolution HPLCMS runs, data collection was done via MassHunter Workstation Software (B.08.00) and data analysis relied on MassHunter Workstation Software Qualitative Analysis (10.0)

DiNicolantonio JJ, Liu J, and OKeefe JH, Magnesium for the prevention and treatment of cardiovascular disease Open Heart vol