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These findings provide substantial support for the neurotrophic hypothesis of depression and specify anatomical and neurochemical targets for potential antidepressant interventions

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[16] In some cases, switching to an alternative agent with different pharmacokinetic properties or discontinuing therapy may be necessary if symptoms significantly impair quality of life or pose health risks

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[1] [3] It slows gastric emptying, increases insulin secretion when blood glucose is elevated, and reduces appetite

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