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Observed molecular effects are dependent on experimental design, concentration, and model system and are used to investigate IGF-1 receptor signalling rather than defined biological or physiological outcomes

KEAP1/NRF2 mutant cells are protected against cysteine-starvation induced ferroptosis due to elevated GCLC expression and limited glutamate accumulation, and dual targeting of cystine import and GCLC activity can overcome the resistance of NRF2 active cells to ferroptosis (Kang et al., Cell Metab 2021)

[27:26] The reason for this weve mentioned that there are 703 amino acids in that molecule

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Sorafenib Combination therapies targeting complementary molecular pathways represent a promising strategy to overcome resistance while potentiating therapeutic efficacy [118]

CMC 19, 40004005