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5 They are crucial players in cell signaling 6 and while detoxifying, specific GST classes, particularly pi and mu, directly interact with signaling molecules like c-Jun N-terminal kinase 1 (JNK1) and apoptosis signal-regulating kinase (ASK1)

The fixed cells were treated with 20 g/mL RNase A and 10 g/mL propidium iodide in PBS at 37 C for 30 min

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From ubiquinol, the electrons pass down the redox gradient through complex III (coenzyme Q-cytochrome c oxidoreductase) to cytochrome c , then to complex IV (cytochrome c oxidase) and to the final acceptor, oxygen (O 2 ) to produce water

These findings establish ferroptosis as a key mechanism in myoglobin-induced nephrotoxicity ( Figure 5 )

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