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We found that biotinylated sv6D and svC1 were poor substrates when assayed immediately after dissolving commercial lyophilized TGM2 in water containing 1 mM EDTA, pH 7.5, and 10 mM dithiothreitol (Figure 5a), but surprisingly, when the solution of TGM2 was stored over several days at 5 C in this medium, the enzyme lost over half of the activity with svL4 but gained the ability to use sv6D and, to a lesser extent, svC1 as substrates (Figure 5b)

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[5] Beyond sodium-channel antagonism, systemic lidocaine exhibits anti-inflammatory and antihyperalgesic properties attributed to inhibition of G-protein-coupled receptors, modulation of NMDA-mediated excitatory transmission, suppression of neutrophil priming, and attenuation of central glial activation

5a) did not reliably quantify any GSS-CAM-TPP