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A common autoimmune-mediated disease of the CNS that is characterized by multiple acute inflammatory foci involving immune-mediated demyelination that can undergo spontaneous remyelination

The consequences of such differential regulatory process for tolerance are still not well understood but are likely to be the basis for biased agonism for arrestin recruitment

Several studies had already focused on this gut-brain axis

Evidence signal SURMOUNT evidence 8 source-backed citations are connected to this page

10.1007/s10735-011-9376-9 80 KhanT.WaseemR.ZehraZ.AimanA.BhardwajP.AnsariJ.et al (2022)

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