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IE can trigger iron overload, and ferroptosis, a regulated cell death process triggered by iron overload, has the ability to potentially modulate, IE

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Western blot analysis revealed that recombinant CXCL10 treatment sharply increased the levels of p-P65 and IFN in the CXCL10 group, whereas IL18R1 antibody treatment reduced their levels close to those of the control in the 10+18ab group (Fig

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