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Argello JM, Raimunda D, Gonzlez-Guerrero M

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Recent work suggested that the reduction in both food intake and body weight observed in response to combined treatment with GLP-1RAs and PYY 3-36 results in synergistically-enhanced activation of discrete hypothalamic and brainstem circuits that regulate appetite, including the arcuate nucleus (ARC), the paraventricular nuclei of the hypothalamus (PVNs), the central nucleus of the amygdala (CeA), and the hindbrain (AP/NTS) 10,12,21,44

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These imbalances were seen to be related to both the increase of pro-inflammatory cytokines (TNF-alpha and IL6) and to the alteration of mechanisms that prevent bacterial translocation, testifying an important role of these microbiological alterations in the evolution of liver disease [44]

Factors such as metabolism and dosing affect its longevity