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Berg et al

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Disrupted gut microbiota may lead to increased intestinal permeability (leaky gut), molecular mimicry (e.g., cross-reactivity between bacterial antigens and TPO), and activation of Th17/IL-17 pathways

A trial of the small molecule inhibitor of -synuclein aggregation minzasolmin also failed to detect differences from placebo on the primary outcome of change from baseline in MDS-UPDRS scores [205, 206]