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In stroke, reduced mitophagy and autophagy may hinder the prompt clearance of damaged mitochondria in the brain, leading to a loss of protection from increased reactive oxygen species (ROS) and reduced mitochondrial ATP supply (Frugier et al., 2015)

However, the long-term trajectory of GLP-1 RArelated costs, including potential sustained increases in drug costs and any unforeseen long-term adverse event management, remains an area requiring further investigation

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