The anti-inflammatory actions of MUFAs are based on their ability to counteract the effects of long-chain saturated fatty acids on hepatocytes, which include reducing endoplasmic reticulum stress, restricting lipotoxicity induced by accumulation of saturated fatty acids, decreasing ROS production, and inhibiting nuclear factor-B (NF-kB) transcription factors by binding peroxisome proliferator-activated receptor (PPAR) and G-protein coupled surface receptor 120 (GPR120) (138)
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It would be interesting to understand what actually maintains that state
Conversely, reducing intracellular copper levels can diminish the synthesis of the copper-dependent enzyme SOD1, thereby substantially impairing the organism's antioxidant capacity
designed an eco-friendly antioxidant nanoplatform based on polyphenol-enriched grape pomace extracts encapsulated within surface-functionalized liposomes that exhibited enhanced BBB permeability
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