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179,180 Nonetheless, through the integration of multi-omics analysis and validation both in vitro and in vivo in NSCLC, a recent study disclosed that intracellular lactate drives extracellular lipolysis and FFA release via non-histone lactylation of apolipoprotein C-II (APOC2), bringing about immunotherapy resistance

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The peptide's exceptional selectivity for senescent cells, combined with its well-characterized mechanism of FOXO4-p53 interaction disruption, provides researchers with powerful capabilities for investigating senescence biology that were previously impossible to study

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