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While this explanation touches on some interesting concepts, there are several points that need clarification and might be oversimplified or speculative: Lets start with neuronal compensation

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EAAC1 is tightly regulated by molecules, including glutamate transporter-associated protein 318 (GTRAP3-18) and miR-96-5p, offering potential therapeutic targets to optimize neuronal GSH levels and mitigate early neurodegeneration (6)

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