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We also demonstrate the utility of HRMS discovery-based approach (sometimes referred to as pharmacometabolomics in clinical applications ( 35, 36 )) to advance a step forward towards precision and personalized medicine and uncover new metabolites markers that can be used to provide better typing of patients related to the kinetic of elimination of a drug or to identify inter-individual susceptibility to develop drug-induced liver injury, as it is the case here with APAP

It is a way to match mechanisms to the clinical picture when fundamentals are already in place

10.1097/WNR.0000000000000046 96

As MPT and JNK are key players in APAP-mediated acute liver failure, it is unclear whether mitochondrial dysfunction and JNK are engaged in low dose APAP exposure without liver injury

"Ultimately, GLP-1 medications are powerful tools, but they require a thoughtful, individualized approach

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