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Treatment-specific, transient elevations in selected cytokines, together with accelerated epithelial wound healing in vitro , support a dual mode of action: barrier stabilization coupled with an adaptive pro-repair immune response

In addition, dysregulated -KG flux from normal reductive anabolism via the TCA cycle toward R-2-HG production has been associated with other metabolic flux impairments and disrupted redox balance 119,120 (Fig

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GRPP: glicentin-related pancreatic polypeptide

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