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These findings highlighted the role of aberrant tumor angiogenesis in generating interstitial hypertension due to leaky, immature blood vessels and impaired lymphatic drainage

Br J Cancer 111 , 6167 (2014)

In cell-based functional assays, AMG 133 shows antagonist activity against human, cynomolgus monkey and rat GIPR (Fig

To further elucidate the therapeutic mechanisms underlying D-CuPs multivalent interactions, comprehensive proteomic analysis revealed two pivotal transcriptional regulators, the tumor suppressor protein p53 and histone deacetylase 7 (HDAC7) to be potential mechanistic targets

Combination therapies (e.g., GLP-1 with GIP or glucagon receptor agonists) and next-generation analogues are being designed to enhance efficacy, tissue targeting, and tolerability

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