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Furthermore, Doxorubicin and Cyclophosphamide reduced synaptic plasticity via hindering expression of brain-derived neurotrophic factor, phosphorylated cyclase response element binding protein, synaptophysin, and postsynaptic density protein 95 whereas protein expression of such synaptic plasticity biomarkers was enhanced by L-carnitine treatment

Nonetheless, it had no impact on the body's natural glucose production, suggesting that the activation of MC3R and MC4R receptors in the central nervous system affects insulin sensitivity in a tissue-specific manner, independently [2]

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