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Note: these studies describe experimental endpoints and do not constitute medical claims

Its balanced potency and consistent results make it a part of many treatment protocols

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In the synovial fluid of individuals with OA, increased lactate facilitates lactylation of UDP-glucose dehydrogenase (UGDH) at lysine 6, disrupting its interaction with signal transducer and activator of transcription 1 (STAT1), suppressing its enzymatic activity and glycosaminoglycan synthesis, and altering its nuclear-cytoplasmic distribution, activating the MAPK signaling pathway and ultimately contributing to OA progression

Prescription DPP-4 inhibitors like sitagliptin extend GLP-1 half-life by blocking this enzyme, approximately doubling circulating GLP-1 levels

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